ENCEFALITE LIMBICA PDF

Lo stesso argomento in dettaglio: Ippocampo anatomia. Con CA4 si fa riferimento al giro dentato. Fornice[ modifica modifica wikitesto ] Lo stesso argomento in dettaglio: Fornice cervello. Continuando anteriormente al talamo si formano le colonne del fornice due fasci simmetrici che si portano in basso: il passaggio davanti al polo anteriore del talamo delimita un foro, il forame interventricolare, che permette la comunicazione fra III ventricolo e ventricolo laterale. Le fibre trasversali sono presenti nel psalterium o lira [7] o commessura del fornice [8] compreso fra le due gambe prima che si fondano per formare il corpo facendo del fornice la formazione commessurale del sistema limbico [7]. Tali nuclei derivano dallo stiramento della sostanza grigia verso la base del proencefalo.

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Sonia Maria Dozzi Brucki. Email: rb. This is an Open Access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Abstract Autoimmune limbic encephalitis ALE associated with systemic lupus erythematosus SLE is a rare entity with few reports in the literature to date.

In general, ALE associated with SLE has a satisfactory response to immunosuppressive treatment RIT , but the pathogenesis of this association is poorly understood and may include an autoimmunity component. We report a case study describing the diagnosis and management of limbic encephalitis in a patient with active Systemic Lupus Erythematosus disease SLE and past medical history of cancer endometrial adenocarcinoma in and papillary urothelial carcinoma in with curative treatment , followed over a one-year period.

We discuss the possible association between limbic encephalitis and all past neoplastic and immune-mediated conditions of this patient. In this particularly case, autoimmunity was the most relevant factor associated with limbic encephalitis given negative neoplastic screening. Moreover, a good response was observed to immunotherapy, not seen with paraneoplastic limbic encephalitis, which is associated with poor response.

In this case, the association of ALE with SLE is possible, since laboratory testing disclosed lupic activity and the patient had involvement of other systems such as hematologic during the period. However, the presence of other surface membrane antibodies are possible in the search for alternative etiologies.

In addition, there may be different degrees of involvement in extra-limbic-system tissues such as the cerebellum, brainstem and thalamus.

Although it is still considered a classical paraneoplastic syndrome, its association with autoimmune disease has been extensively studied. For instance, some patients previously thought to have viral encephalitis will be found to have a treatable autoimmune disease. Therefore, there is a lack of understanding on the frequency and power of this association. These symptoms were followed by anterograde amnesia and temporal disorientation initiated 3 days before the hospitalization.

Clinical and neurologic examination was normal except for temporal disorientation, low scores on the Mini-Mental State Examination and episodic memory impairment Table 1.

Table 1 Cognitive performance on baseline and follow-up. Cognitive Assessment Follow-up.

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Sintomi, tipi e cause di encefalite limbica

Tutto su scienza, cultura, educazione, psicologia e stile di vita. Questa patologia si verifica a causa del coinvolgimento della regione mediale dei lobi temporali. Fonte: groups. In questo articolo esaminiamo le principali caratteristiche di questa malattia. Le principali caratteristiche cliniche EL sono la perdita subacuta di memoria a breve termine, lo sviluppo di una sindrome demenza e coinvolgimento infiammatorio della sostanza grigia limbic in associazione con carcinoma bronchiale.

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Il Giornale Italiano di Radiologia Medica 2019 Marzo-Aprile;6(2):119-27

You are on page 1of 50 Search inside document Elda A Botelho Definio Desordens neurolgicas associadas a neoplasias sistmicas, causadas por mecanismos outros que metstases, causas metablicas, deficincias nutricionais, infeces, coagulopatias ou efeitos colaterais do tratamento do cncer. Citologia negativa na ausncia de impregnao menngea na RM exclui carcinomatose menngea Alteraes inflamatrias pleocitose, sntese intratecal de IgG, bandas oligoclonais confirmam a presena de alterao neurologica inflamatria ou inumomediada. No melhora com plasmaferese. Melhora com o tratamento do tumor e transplante de medula ssea. CA aparece antes ou meses aps a SGB Tratamento: plasmaferese, IgIV Resposta teraputica semelhante aos casos sem TU Neuropatia autonmica paraneoplsica Acompanha outras sd paraneoplsicas encefalomielite, neuropatia sensorial Hipotermia, hipoventilao, apnia do sono, gastroparesia, pseudo-obstruo intestinal, arritmias cardacas morte subta , diaforese oat cell anti-Hu , CA pncreas, tireide e reto, linfoma Hodgkin, TU carcinide do pulmo anti receptor de acetilcolina Pouca melhor com o tratamento do TU ou imunoterapia prednisona, micofenolato mofetil Vasculite de nervo perifrico paraneoplsica Dor, fraqueza proximal e dficit sensoriomotor simtrico lembrando mononeurite mltipla, Bipsia: infiltrado de predomnio mononuclear em pequenas veias, sem necrose ou clulas malignas Geralmente precede a descoberta do TU. Pode desaparecer com bloqueio da juno neuromuscular.

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